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BPC-157 reportedly induced increased levels of phosphorylation of both PAK and paxillin, while the total protein level remained unchanged
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It seems that the reason carnitine was unable to increase muscle carnitine concentration in these studies for 2 reasons: Poor bioavailability (only 20% for of a 2-6 g dose is absorbed) and Because the transport of carnitine into the muscle was limited
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Reframing the conceptual paradigm from a reductionist focus on liver flushing toward the targeted support and optimization of glutathione-dependent detoxification pathways establishes a model primarily oriented around the promotion and maintenance of cellular resilience
It is entirely valid to feel frustrated by the lack of simple answers
